Proteostasis and Proteolysis (Record no. 93082)

MARC details
000 -LEADER
fixed length control field 04208namaa2200481uu 4500
001 - CONTROL NUMBER
control field doab100138
003 - CONTROL NUMBER IDENTIFIER
control field oapen
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20260305123951.0
006 - FIXED-LENGTH DATA ELEMENTS--ADDITIONAL MATERIAL CHARACTERISTICS
fixed length control field m o d
007 - PHYSICAL DESCRIPTION FIXED FIELD--GENERAL INFORMATION
fixed length control field cr|mn|---annan
008 - FIXED-LENGTH DATA ELEMENTS--GENERAL INFORMATION
fixed length control field 230512s2022 xx |||||o ||| 0|eng d
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9780367499327
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9780367499334
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9781003048138
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9781003048138
024 7# - OTHER STANDARD IDENTIFIER
Standard number or code 10.1201/9781003048138
Source of number or code doi
040 ## - CATALOGING SOURCE
Original cataloging agency oapen
Transcribing agency oapen
041 0# - LANGUAGE CODE
Language code of text/sound track or separate title eng
042 ## - AUTHENTICATION CODE
Authentication code dc
245 00 - TITLE STATEMENT
Title Proteostasis and Proteolysis
260 ## - PUBLICATION, DISTRIBUTION, ETC.
Name of publisher, distributor, etc. Taylor & Francis
Date of publication, distribution, etc. 2022
300 ## - PHYSICAL DESCRIPTION
Extent 1 online resource
336 ## - CONTENT TYPE
Content type term text
Content type code txt
Source rdacontent
337 ## - MEDIA TYPE
Media type term computer
Media type code c
Source rdamedia
338 ## - CARRIER TYPE
Carrier type term online resource
Carrier type code cr
Source rdacarrier
506 0# - RESTRICTIONS ON ACCESS NOTE
Terms governing access Free-to-read
Standardized terminology for access restriction Unrestricted online access
Source of term star
520 ## - SUMMARY, ETC.
Summary, etc. Proteins are the key decision-making workforce for practically all cellular signals. The precise equilibrium between protein translation, folding, function and timely degradation, also known as "proteostasis", determine cellular health as well as organismal maintenance and survival. Loss of cellular proteostasis is linked with physiological processes of deterioration such as aging, with conditions characterized by supraphysiological oxidative stress as well as with illness, including age-related diseases, neurodegeneration, inflammation, dystrophies and cancer. This book includes 18 chapters logically organized to allow comprehensive understanding of how maintenance of proteostasis protects cellular and organismal health, and how environmental and metabolic pressure can impair proteostasis and lead to disequilibrium and disease. Each chapter contains up-to-date information on its respective topic while some of them review the interplay of certain proteostatic mechanisms, a newly arising topic. Importantly, most chapters include tangible examples of how failure of proteostasis can underlie aging and disease. We hope that the compilation of the above topics will assist both novice and experienced researchers and students to become more familiar with the subject of proteostasis. In the long run, we hope that this book will inspire its readers and eventually promote new ideas and new research studies.
540 ## - TERMS GOVERNING USE AND REPRODUCTION NOTE
Terms governing use and reproduction All rights reserved
Uniform Resource Identifier <a href="http://oapen.org/content/about-rights">http://oapen.org/content/about-rights</a>
546 ## - LANGUAGE NOTE
Language note English
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term ad, age, alejandro, alihamze, anna, arantxa, aspects, assembly, Athanasopouloi, autophagy, between, cell, central, chaerone, chapter, Chondrogianni, complex, concluding, conculsions, contents, control, crosstalk, current, deciphering, definition, degradation, Diana, diseases, during, Efstathios, elF5A, Elah, Elisabeth, Factors, Farras, fathinajafabadi, Ferrando, Ferriz, folding, functions, future, gioran, gonos, heat, heidi, integrity, interplay, introsuction, irreversible, judith, kapetanou, kinetic, leading, localized, Marianna, Martinez, mechanisms, MicroRNAs, misfolding, molecular, motifs, network, Kiki, Olzscha, organellar, other, oxidative, panfilova,pathway, pauses, perspectives, pick, polu, pro, process, Proteasome, protein, proteinopathies, proteolysis, proteome, proteostasis, proteostatic, quality, references, regulation, regulators, related, remarks, rescues, response, reversible, ribosomal, riemer, rosa, RQC, S, shock,significance, sophia, stalling, stress, system, systemic, systems, table, targeting, term, thermodynamic, transcriptional , translation, translational, treatments, ubiquitin, unfolded
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term thema EDItEUR::M Medicine and Nursing::MF Pre-clinical medicine: basic sciences
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term thema EDItEUR::P Mathematics and Science::PN Chemistry::PNN Organic chemistry
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term thema EDItEUR::P Mathematics and Science::PS Biology, life sciences
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSD Molecular biology
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name Chondrogianni, Niki
Relationship edt
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name Chondrogianni, Niki
Relationship oth
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name Gioran, Anna
Relationship edt
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name Gioran, Anna
Relationship oth
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name PICK, ELAH
Relationship edt
720 1# - ADDED ENTRY--UNCONTROLLED NAME
Name PICK, ELAH
Relationship oth
793 0# -
-- DOAB Library.
856 40 - ELECTRONIC LOCATION AND ACCESS
Uniform Resource Identifier <a href="https://directory.doabooks.org/handle/20.500.12854/100138">https://directory.doabooks.org/handle/20.500.12854/100138</a>
Access status 0
Public note Free-to-read: DOAB: description of the publication

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